By Juergen Kraemer, Jürgen Krämer
The recent variation of this booklet offers the present details at the diagnostic options and healing methods that would support clinicians optimize take care of ...
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Extra resources for Intervertebral disk diseases: causes, diagnosis, treatment, and prophylaxis
1992), and Brinkmann (2004). Radiologically measurable increases in disk height after traction have been described by many groups (Krusen and McFarlane 1943, De Sèze and Levernieux 1952, Fraser 1954, Crisp 1955, Judovich 1955, Lawson and Godfrey 1958, Lehmann 1958, Cyriax 1959, Worden and Humphrey 1964, Mathews 1968, Colachis 1969, Krämer 1973, Eklund and Corlett 1984, Krag et al. 1985, Ledsome 1996). In accordance with the pressure-dependent fluid shifts described above, increased compressive stress on the disks, e.
Paucity of movement also harms the nutritive state of the intervertebral tissues. The intervertebral disk is avascular in adults and obtains nutrients by diffusion through an osmotic system. The existence of pressure-dependent fluid shifts within the disk has also been confirmed by recent studies. A regular alternation of mechanical stress and relaxation aids intradiscal metabolism, but a constantly maintained posture—particularly one in which the spine is under stress—impairs it. All changes in the anterior portion of the motion segment affect the intervertebral joints as well.
The mucopolysaccharides lend the matrix its elasticity and viscosity through their ability to bind large amounts of water. Mucopolysaccharides and other macromolecules are synthesized partly intracellularly, partly extracellularly. Amino sugars and other building blocks of the acidic mucopolysaccharides are formed intracellularly as intermediate products of glucose metabolism. The cartilage cells are the most important sites of metabolic activity within the disk. They synthesize their own organic matrix, which consists of collagen and a mucopolysaccharide–protein complex.