Physical Medicine Rehabilitation

Atlas of Neuromuscular Diseases: A Practical Guideline by Eva L. Feldman

By Eva L. Feldman

The atlas encompasses a finished define of neuromuscular ailments, written through skilled American and ecu authors. It discusses all features of neuromuscular problems together with the cranial nerves, spinal nerves, motor neurone disorder, the nerve plexus, and so forth. each one bankruptcy is uniformly based into anatomy, indicators, indicators, pathogenetic chances, prognosis and differential prognosis, remedy and analysis. also, the diagnostic instruments and investigations utilized in neuromuscular disorder are defined and a realistic consultant is given on easy methods to develop from indicators to syndromes. for every ailment the healing strategies are defined. It includes huge variety of medical and histologic images and artists drawings.

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Extra info for Atlas of Neuromuscular Diseases: A Practical Guideline

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Thieme, Stuttgart, pp 11–53 Wilson-Pauwels L, Akesson EJ, Stewart PA (1988) Cranial nerves. Anatomy and clinical comments. Decker, Toronto Philadelphia 39 Oculomotor nerve Genetic testing NCV/EMG Laboratory Imaging (PNP: NCV) + (Diabetes) ++ Biopsy Lee screen + Fig. 2. 1 Oculomotor nerve, 2 Abducens nerve, 3 Trochlear nerve, 4 Cross section through brainstem, 5 Internal carotid artery Fig. 3. Oculomotor nerve paresis: A Complete ptosis; B Upon lifting of the lid lateral deviation of left bulbus.

Therapy The recovery rate over 6 months was observed to be higher in cases of diabetic etiology than other non-selected cases. Prognosis Berlit P (1991) Isolated and combined pareses of cranial nerves III, IV, and VI. A retrospective study of 412 patients. J Neurol Sci 103: 10–15 Jacobson DM, Marshfield DI, Moster ML, et al (2000) Isolated trochlear nerve palsy in patients with multiple sclerosis. Neurology 55: 321–322 Keane JR (1993) Fourth nerve palsy: historical review and study of 215 inpatients.

In LEMS, a presynaptic disorder, calcium channel autoantibodies directed against the P/Q type channels have been described. These autoantibodies are Autoimmune testing in neuromuscular transmission and muscle disorders 26 detected in nearly 100% of patients with LEMS. Antibodies against the N-type channel are detected in 74% of LEMS patients. Neuronal acetylcholine receptor antibodies are directed against AchR in autonomic ganglia, resulting in autonomic dysfunction. Patients with MG or LEMS have a higher association with other autoantibodies, like thyroid peroxidase, thyreoglobulin, gastric parietal cell, and glutamic acid decarboxylase (GAD).

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