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Atherosclerosis III: Proceedings of the Third International by E. B. Smith, S. Björkerud, R. H. More, D. Sinapius, G. S.

By E. B. Smith, S. Björkerud, R. H. More, D. Sinapius, G. S. Berenson, W. Insull (auth.), G. Schettler, A. Weizel (eds.)

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The only relationship that we found in the fatty streaks, for example, was in lysolecithin in work that Dr. Lang did several years ago. When we repeated it on a more recent group of fatty streak preparations, we could not confirm this correlation. Therefore, I feel that this relationship may just have been a factitious observation. One other observation in the same general area is that one can make models of these inclusions and can do this with pure cholesterol esters in which we have no phospholipid.

They are there, and unless you stain for them very specifically, you are not very aware that they are there. Dr. Finlayson: Dr. Berenson, may I ask you, have you examined mucopolysaccharides in tendons. If you have, what do they share in common with mucopolysaccharides of the aorta? The thing they share in common, I think will help us to elucidate why lipoproteins are sticky. 10 Dr. of the xanthemateus material. We have net analysed tendons. Dr. Hodara: I would like te make some comments en Dr.

Illustration of some functional and pathophysiological concepts related to arterial endothelium This type of lesion will eventually be reendothelialized in the normo-lipidemic rabbit (Fig. 3, II D). e. foam cells occur. From general tissue biological criteria, this tissue is more or less necrotic (Fig. 3, II D, black area). In contrast to the abundance of lipids as seen in sections, the actual cholesterol content its considerably lower and the proportion of cholesterol ester is also lower than in the preceding stage (Bondjers and Bjorkerud, 1973b; Bjorkerud and Bondjers, 1973b).

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